Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes

Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes

Amyotrophic lateral sclerosis (ALS) is an incurable and lethal neurodegenerative disease in which progressive motor neuron loss and associated inflammation represent major pathology hallmarks. Both the prevention of neuronal loss and neuro-destructive inflammation are still unmet challenges. Medi...

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Autoren: Bette, Michael, Cors, Eileen, Kresse, Carolin, Schütz, Burkhard
Formato: Artigo
Idioma:inglés
Publicado: Philipps-Universität Marburg 2022
Schlagworte:
neurodegeneration
adaptive immune system
amyotrophic
monocyte
Medizin
ozone
neuroinflammation
Medical sciences Medicine
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Número de Clasificación: urn:nbn:de:hebis:04-es2022-01780
Data de Publicación: 2022-11-23
Fonte: Erstveröffentlichung: Bette, M.; Cors, E.; Kresse, C.; Schütz, B. Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes. Int. J. Mol. Sci. 2022, 23, 3403. https://doi.org/10.3390/ijms23063403
Downloads: 23 (2024), 19 (2023), 1 (2022)
Lizenz: https://creativecommons.org/licenses/by/4.0
URL de acceso: https://archiv.ub.uni-marburg.de/es/2022/0178
https://doi.org/10.3390/ijms23063403

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