Analysis of the molecular mechanisms of prestin-mediated cochlear amplification via a cysteine accessibility study

Analysis of the molecular mechanisms of prestin-mediated cochlear amplification via a cysteine accessibility study

Prestin is the protein being responsible for electromotility in mammals. It is a member of the SLC26 (Solute Carrier) family, which function as anion transporters. In contrast to the other members, mammalian prestin does not function as an anion transporter but produces electromotility. Non-mammalia...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijä: Hartmann, Julia
Muut tekijät: Oliver, Dominik (Prof. Dr.) (BetreuerIn (Doktorarbeit))
Aineistotyyppi: Dissertation
Kieli:englanti
Julkaistu: Philipps-Universität Marburg 2021
Aiheet:
electromotility
nicht-lineare Kapazität
NLC
SCAM
Rattenprestin
non-linear capacity
Cystein Zugänglichkeitsstudie
prestin
zebrafish prestin
Medizin
cochlear amplification
Kochleäre Amplifikation
Elektromotilität
molecu
Prestin
rat prestin
cysteine accessibility study
Zebrabärblingpre
Medical sciences Medicine
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