The psychiatric risk gene Cacna1c regulates mitochondrial function in cellular stress responses

The psychiatric risk gene Cacna1c regulates mitochondrial function in cellular stress responses

Affective disorders such as major depression and bipolar disorder are among the most prevalent forms of mental illness, and their pathophysiology involves complex interactions between genetic and environmental risk factors. However, the underlying mechanisms explaining how genetic and environmental...

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Autore principale: Michels, Susanne
Altri autori: Culmsee, Carsten (Prof. Dr.) (Relatore della tesi)
Natura: Dissertation
Lingua:inglese
Pubblicazione: Philipps-Universität Marburg 2018
Soggetti:
Oxidativer Stress
Gen-Umwelt Interaktionen
Cav1.2 Neuroinflammation Affective disorders Mitochondria Gene-environment interactions Oxidative stress
Affektive Störungen
Cacna1c
Pharmakologie, Therapeutik
Mitochondrien
Pharmacology & therapeutics, prescription drugs
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