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MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S
Mutations in MAGED2 cause transient Bartter syndrome characterized by severe renal salt wasting in fetuses and infants, which leads to massive polyhydramnios causing preterm labor, extreme prematurity and perinatal death. Notably, this condition resolves spontaneously in parallel with developmental...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | 論文 |
| 言語: | 英語 |
| 出版事項: |
Philipps-Universität Marburg
2022
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| 主題: | |
| オンライン･アクセス: | PDFフルテキスト |
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PDFフルテキスト| 請求記号: |
urn:nbn:de:hebis:04-es2023-00501 |
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| 出版日付: |
2023-05-16 |
| ソース: |
Erstveröffentlichung: Seaayfan, E.; Nasrah, S.; Quell, L.; Kleim, M.;Weber, S.; Meyer, H.; Laghmani, K.; Kömhoff, M. MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S. Cells 2022, 11, 2546. https://doi.org/10.3390/cells11162546 |
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27 (2024), 63 (2023) |
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https://creativecommons.org/licenses/by/4.0 |
| アクセスURL: |
https://archiv.ub.uni-marburg.de/es/2023/0050 https://doi.org/10.3390/cells11162546 |