MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S

MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S

Mutations in MAGED2 cause transient Bartter syndrome characterized by severe renal salt wasting in fetuses and infants, which leads to massive polyhydramnios causing preterm labor, extreme prematurity and perinatal death. Notably, this condition resolves spontaneously in parallel with developmental...

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Bibliografski detalji
Glavni autori: Seaayfan, Elie, Nasrah, Sadiq, Quell, Lea, Kleim, Maja, Weber, Stefanie, Meyer, Hemmo, Laghmani, Kamel, Kömhoff, Martin
Format: Članak
Jezik:engleski
Izdano: Philipps-Universität Marburg 2022
Teme:
MAGED2
Medizin
G-alpha-S
MDM2
Bartter
hypoxia
Medical sciences Medicine
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Signatura: urn:nbn:de:hebis:04-es2023-00501
Datum izdanja: 2023-05-16
Izvor: Erstveröffentlichung: Seaayfan, E.; Nasrah, S.; Quell, L.; Kleim, M.;Weber, S.; Meyer, H.; Laghmani, K.; Kömhoff, M. MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S. Cells 2022, 11, 2546. https://doi.org/10.3390/cells11162546
Downloads: 27 (2024), 63 (2023)
Lizenz: https://creativecommons.org/licenses/by/4.0
URL pristupa: https://archiv.ub.uni-marburg.de/es/2023/0050
https://doi.org/10.3390/cells11162546

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