MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S
Mutations in MAGED2 cause transient Bartter syndrome characterized by severe renal salt wasting in fetuses and infants, which leads to massive polyhydramnios causing preterm labor, extreme prematurity and perinatal death. Notably, this condition resolves spontaneously in parallel with developmental...
Guardat en:
Autors principals: | , , , , , , , |
---|---|
Format: | Article |
Idioma: | anglès |
Publicat: |
Philipps-Universität Marburg
2022
|
Matèries: | |
Accés en línia: | PDF a text complet |
Etiquetes: |
Afegir etiqueta
Sense etiquetes, Sigues el primer a etiquetar aquest registre!
|
Internet
PDF a text completSignatura: |
urn:nbn:de:hebis:04-es2023-00501 |
---|---|
Data de publicació: |
2023-05-16 |
Font: |
Erstveröffentlichung: Seaayfan, E.; Nasrah, S.; Quell, L.; Kleim, M.;Weber, S.; Meyer, H.; Laghmani, K.; Kömhoff, M. MAGED2 Is Required under Hypoxia for cAMP Signaling by Inhibiting MDM2-Dependent Endocytosis of G-Alpha-S. Cells 2022, 11, 2546. https://doi.org/10.3390/cells11162546 |
Downloads: |
27 (2024), 63 (2023) |
Lizenz: |
https://creativecommons.org/licenses/by/4.0 |
URL d'accés: |
https://archiv.ub.uni-marburg.de/es/2023/0050 https://doi.org/10.3390/cells11162546 |